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. 2018 Feb 15;9(3):283. doi: 10.1038/s41419-018-0287-5

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a WB results show the knocking down efficiency of EphA2 in Huh7 and LM3 cells. b Knocking down EphA2 suppresses Huh7 cells invasion. c Knocking down EphA2 suppresses LM3 cells invasion. d WB results show the efficiency of the disruption of EphA2 expression by transfecting with siEphA2 in Huh7-shTR₄ cells. e Interrupting EphA2 can partially reverse the increasing migration in Huh7-shTR₄ cells. f Interrupting EphA2 can partially reverse the increasing invasion in Huh7-shTR₄ cells. g WB results show the efficiency of the disruption of EphA2 expression by transfecting with siEphA2 in LM3-shTR₄ cells. h Interrupting EphA2 can partially reverse the increasing migration in LM3-shTR₄ cells. i Interrupting EphA2 can partially reverse the increasing invasion in LM3-shTR₄ cells. (**P < 0.01, ***P < 0.001)