Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2018 Feb 21;9(3):303. doi: 10.1038/s41419-018-0291-9

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

Fig. 4 — a, b NFYA and SATB1 were overexpressed in endometrial cancer tissues, as shown by qRT-PCR and western blot (33 cases in cancer group and 20 cases in normal group). c The predicted miR-302a-3p/miR-3130-3p binding sites in NFYA and the designed mutant sequence are indicated. A luciferase assay of miR-302a-3p/miR-3130-3p and NFYA was conducted. d1, d2 The results of qRT-PCR and western blot demonstrated that LINC01016 regulated NFYA and SATB1 expression in Ishikawa and RL-95-2 cells. e1, e2 Findings of qRT-PCR and western blot showed that miR-302a-3p/miR-3130-3p regulated NFYA and SATB1 expression in Ishikawa and RL-95-2 cells. Data are presented as the means ± SD from three independent experiments. *P < 0.05

Fig. 4 — a, b NFYA and SATB1 were overexpressed in endometrial cancer tissues, as shown by qRT-PCR and western blot (33 cases in cancer group and 20 cases in normal group). c The predicted miR-302a-3p/miR-3130-3p binding sites in NFYA and the designed mutant sequence are indicated. A luciferase assay of miR-302a-3p/miR-3130-3p and NFYA was conducted. d1, d2 The results of qRT-PCR and western blot demonstrated that LINC01016 regulated NFYA and SATB1 expression in Ishikawa and RL-95-2 cells. e1, e2 Findings of qRT-PCR and western blot showed that miR-302a-3p/miR-3130-3p regulated NFYA and SATB1 expression in Ishikawa and RL-95-2 cells. Data are presented as the means ± SD from three independent experiments. *P < 0.05