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. 2018 Feb 20;9(3):292. doi: 10.1038/s41419-017-0253-7

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a CaM protein levels were higher in GBM (n = 3) than in lower-grade glioma (WHO Grade II: n = 3, WHO Grade III: n = 3) and normal tissues (n = 3), as determined by western blot. b CaM expression was increased in GBM samples (n = 14) compared to lower-grade glioma (WHO Grade II: n = 9, WHO Grade III: n = 12) and normal brain tissues (n = 6) using immunohistochemical staining (Data are presented as means ± SDs of three different microscopic visions from every independent experiment, *p < 0.05, **p < 0.01, ****p < 0.0001). c Five GBM patients’ MRI images with representative images of immunohistochemical staining of CaM (Images were taken from three different microscopic visions from every independent experiment, Red triangle: disseminated tumor)