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. 2018 Jan 26;9(2):130. doi: 10.1038/s41419-017-0131-3

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Tumors formed in nude mice. SK-hep-1 cells stably overexpressing miR-874 or empty vector were injected into the flanks of nude mice, and the mice were killed after 5 weeks. b Surgically removed tumor tissues from nude mice 5 weeks post-inoculation. c Tumor volume at different time points after inoculation. d Histological images of resected tumors showed that tumor tissue consisted of HCC cells. e Postulated mechanism of miR-874 inhibiting proliferation and metastasis of HCC through DOR–EGFR–ERK pathways. Data are shown as the mean ± SD (*p < 0.05)