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. 2018 Feb 14;9(2):240. doi: 10.1038/s41419-018-0296-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 9 — Retinal homogenates (5.0 µg protein) from three independent wild-type and rod-cre PKM2 KO mice were subjected to immunoblot analysis with anti-opsin a, anti-arrestin b, anti-Tα c, anti-PDE6β d, anti-PDEγ e, anti-CNGA1 f, anti-RGS9-1 g, anti-Gβ5L/S h, anti-R9AP i, anti-M-opsin j, anti-cone-arrestin k, and anti-actin l antibodies. We normalized the protein expression to rod arrestin m and then calculated the ratios (rod-cre PKM2-KO/PKM2-WT). Data presented as a ratio are mean ± SEM (n = 3). **p<0.01, *p < 0.02