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. 2018 Feb 12;9(2):206. doi: 10.1038/s41419-017-0227-9

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Double labeling of synaptophysin and bungarotoxin of symptomatic wild-type mice treated with vehicle (a–c), SOD1 G93A mice treated with vehicle (d–f), SOD1 G93A mice treated with l-BMAA PC (g–i), SOD1 G93A/ncx3+/− mice treated with vehicle (j–l) and SOD1 G93A /ncx3+/− mice treated with l-BMAA PC (m–o). Scale bar 10 µm. Cell counting analysis of gastrocnemius NMJs of wild-type vehicle, SOD1 G93A vehicle, SOD1 G93A PC, SOD1 G93A/ncx3+/− vehicle and SOD1 G93A /ncx3+/− PC. The data are expressed as the percentage of 270 or more NMJs from each group. Data are expressed as mean ± SEM (n = 5 for each group). *P < 0.05 vs. respective wild-type vehicle. **P < 0.05 vs. all other experimental groups. P values were obtained using two-way ANOVA with Bonferroni’s correction for multiple comparisons