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. 2018 Jan 19;9(2):63. doi: 10.1038/s41419-017-0094-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a A IHC score was assigned to the expression level of PPIL2 and SNAI1 in normal/pericarcinomatous breast tissues (n = 5), breast fibroadenoma tissues (n = 9), and ductal breast cancer tissues (n = 34, 20×). b, c The figures showed that the expression of PPIL2 was significantly less in ductal breast cancer tissues. The expression of SNAI1 was higher in ductal breast cancer tissues than the others. *P < 0.05. d There was a negative correlation between the expression of PPIL2 and SNAI1 level in different breast tissues (r = −0.2936 P = 0.0429). e Lower PPIL2 levels were observed in areas where cancer cells appeared to be traversing the basement membrane to the peripheral tissue (10×)