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. 2018 Jan 18;9(2):27. doi: 10.1038/s41419-017-0189-y

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a–h Representative photographs of subcutaneous tumor xenografts and peritoneal dissemination in nude mice. The arrowheads point to tumor nodules or liver metastasis nodes. The subcutaneous tumors in the control group were remarkably larger than those in the overexpression group. The number of peritoneal dissemination metastatic nodules in the control group was significantly higher than that in the PCDHGA9 overexpression group. i Representative images showed liver and kidney metastasis with SGC-7901-Vector and MGC-803-KD cell peritoneal xenografts. j IHC staining analyses show the staining of PCDHGA9 and Ki-67 in subcutaneous tumors. Overexpression of PCDHGA9 significantly weakened Ki-67 staining. Original magnification ×200. (**p < 0.01, ***p < 0.001)