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. 2018 Jan 29;15(4):4390–4396. doi: 10.3892/ol.2018.7900

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Copyright: © Ye et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — Increased miR-30d inhibits proliferation, migration and invasion of SK-ES-1 cells. (A) CCK-8 assay revealed that ectopic miR-30d significantly suppressed cell growth in SK-ES-1 cells (***P<0.001, vs. untreated). Transwell assay (B) images (magnification, ×100) and (C) quantification demonstrated that invasive potential was significantly reduced in the miR-30d mimic group relative to the untreated group (***P<0.001, vs. untreated). Wound healing assay (D) images (magnification, ×40) and (E) quantification showed that the overexpression of miR-30d markedly repressed the migration of SK-ES-1 cells compared with the control group (***P<0.001, vs. untreated). miR, microRNA; CCK-8, Cell Counting Kit-8.