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. 2018 Jan 26;3:3. doi: 10.1038/s41392-017-0003-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — a–b CCK-8 assays were used to evaluate the effect of upregulation of miR-338-5p on Hep3B or Huh7 cell proliferation. c–d Cell proliferation was promoted after transfection with miR-338-5p inhibitors. e–f Cell colony formation assays showed the effects of overexpressing or knocking down miR-338-5p on HCC cell growth. Student’s t-test (two-tailed) was employed to analyze the data. Data in all experiments are presented as the means ± SD of three independent experiments. *P < 0.05, **P < 0.01, ***P < 0.001 vs. NC