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. 2018 Mar 5;8:3973. doi: 10.1038/s41598-018-22462-y

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Dopamine reuptake were delayed by exercise. The kinetics of dopamine signals were determined by measuring the decay rate constant (τ) of evoked dopaminergic signals in brain slices in each group. The dopamine reuptake rate (tau) was lower in the exercise group; the tau value not only for tonic release but also for phasic release which was higher in PD animals. Nomifensine application did not affect the reuptake rate in the tonic phase but prolonged the reuptake rate of phasic release in the PD with exercise group.