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. 2018 Feb 27;10(2):172–185. doi: 10.4254/wjh.v10.i2.172

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©The Author(s) 2018. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article which was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Computed tomography (CT) and magnetic resonance imaging (MRI) of (A, C, E, G, I) glycogenic hepatopathy (GH) in a 13-year-old girl and (B, D, F, H, J) non-alcoholic fatty liver disease (NAFLD) in a 13-year-old boy. On CT, GH was (A) high density, but NAFLD was typically (B) low density; On T2-weighted imaging (T2WI), both enlarged livers were (C) 19.1 cm and (D) 16.8 cm along the right midclavicular line; On gradient dual-echo MRI, the GH liver was iso-intense between the (E) in-phase and (G) out-of-phase images, namely; I: Low intensity on subtraction. The NAFLD liver, however, had low intensity on the (F) in-phase image, and high intensity on the (H) out-of-phase image, namely; J: High intensity on subtraction. With permission from Saikusa et al[22], John Wiley and Sons publications.