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. 2017 Aug 8;9(11):9608–9617. doi: 10.18632/oncotarget.20033

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Copyright: © 2018 Davidson et al.

This is an open-access article distributed under the terms of the Creative Commons Attribution License 3.0 (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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(A) SMARCA4 knockdown in HPDE cells by siRNA, confirmed by western blot, compared to non-targeting control (NTC); GAPDH serves as a loading control. (B) Bar graph summarizing fold-reductions in IC₅₀ with SMARCA4 (vs. NTC) knockdown, for 6 different chemotherapy agents. (C-H) Dose-response curves comparing HPDE cell viability with SMARCA4 (vs. NTC) knockdown, for (C) cisplatin, (D) oxaliplatin, (E) irinotecan, (F) 5-fluoruracil, (G) gemcitabine, and (H) paclitaxel. IC₅₀ values indicated.