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. 2018 Feb 1;15(4):4827–4836. doi: 10.3892/ol.2018.7922

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Copyright: © Fu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — NP-visnagin ameliorated cardiac dysfunction 4 weeks after ischemia/reperfusion. (A) M-mode echocardiography was performed for rats at 4 weeks after intravenous injection with NP-visnagin and other treatments. (B) Effect of NP-visnagin on LVEDD, LVESD, LVEF and LVFS. (C) Systolic blood pressure was detected through the micromanometer catheter method, and the LVSP, the rate of rise of left ventricular pressure (dP/dt) and LVEDP were calculated. (D) Based on Masson staining, the effects of NP-visnagin and the other treatments on fibrosis at 4 weeks after intravenous injection were detected; representative images are shown. (E) Quantification of fibrosis. *P<0.05; **P<0.01; ***P<0.005. NP, nanoparticles; LVEDD, left ventricular end-diastolic dimension; LVESD, left ventricular end-systolic dimension; LVEF, left ventricular ejection fraction; LVFS, left ventricular fractional shortening; LVSP, left ventricular systolic pressure; LVEDP, left ventricular end-diastolic pressure; MI, myocardial infarction.