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. 2018 Jan;13(1):86–93. doi: 10.4103/1673-5374.224373

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Copyright: © Neural Regeneration Research

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 3.0 License, which allows others to remix, tweak, and build upon the work non-commercially, as long as the author is credited and the new creations are licensed under the identical terms.

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Effects of EA pretreatment on cellular apoptosis and neurological deficit scores in rats with focal cerebral ischemia/reperfusion (IR) injury.

(A) Terminal deoxyribonucleotidyl transferase-mediated dUTP-digoxigenin nick end labeling (TUNEL) staining of cells (arrows) at 24 hours after IR or sham surgery. Scale bars: 50 μm. (B) Proportion of TUNEL-positive cells in the ischemic penumbra (n = 4 rats per group). (C) Neurological deficit score at 6, 24 and 48 hours after reperfusion or sham surgery (n = 8 per group at 6 and 48 hours after reperfusion; n = 16 per group at 24 hours after reperfusion). Data are presented as the mean ± SD, and analyzed by one-way analysis of variance followed by the post hoc Student-Newman-Keuls test. **P < 0.01, vs. sham group; #P < 0.05, vs. electroacupuncture (EA) group at the same time point. I: Sham; II: EA 6 hours; III: IR 6 hours; IV: EA 24 hours; V: IR 24 hours; VI: EA 48 hours; VII: IR 48 hours.