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. 2017 Dec 18;12(1):231–243. doi: 10.1007/s12079-017-0440-4

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© The Author(s) 2017

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 3 — An enhancer at -135 kb upstream of CCN2 drives transgene activity within articular chondrocytes. Strong transgene expression was observed within the proximity of synovial joints (more specifically the elbow (c), wrist (d), femoral aspect of the knee (f) ankle (g) and hind limb paw (h)). Hypertrophic chondrocytes, such as in the vicinity of the primary ossification centres of radius and ulna (c) in addition to the hip (e) and femur (f) were not stained. β-galactosidase activity was absent within all other tissues including the chondrocranium (a) and ribs (b) (Bar = 100 μm)