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. 2018 Mar 10;12:63. doi: 10.1186/s13256-018-1588-0

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — a A large area with multiacinar necrosis accompanied by hemorrhage and parenchymal collapse (stars). This resulted in liver shrinkage and wrinkling (arrows) of the Glisson’s capsule (hematoxylin and eosin stain, original magnification ×100). b Architectural distortion with bridging necrosis (arrows) linking central and portal zones, collapse, and hepatocellular regeneration (hematoxylin and eosin stain, original magnification ×50). c Periportal necrosis and central portal bridging (star), prominent ductular reaction (arrows), loose connective tissue stroma with liver cell loss, inflammation, and clusters of pigment-laden macrophages (hematoxylin and eosin stain, original magnification ×100). d This portal tract (on the right side of the image) is expanded by significant mononuclear inflammatory infiltrate (star) and ductular structures (whose cells have a variable morphology including intermediate forms). A central vein shows venulitis (arrow) and is located adjacent to the periportal zone because of panacinar necrosis (hematoxylin and eosin stain, original magnification ×200)