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. 2018 Jan 12;9(16):12982–12994. doi: 10.18632/oncotarget.24204

Figure 3. OA increased [Ca2+]i and activated the PI3K/Akt signaling pathway in a CD36-dependent manner.

Figure 3

(A) The changes in [Ca2+]i in response to OA and/or CD36 siRNA in HC11 cells. (B) Western blot analysis of p-PI3K, PI3K, p-Akt, and Akt in HC11 after a 4-day culture in the presence of OA and/or CD36 siRNA. β-actin was used as the loading control. (C) Mean ± SEM of immunoblotting bands of p-PI3K/PI3K and p-Akt/Akt, and the intensities of the bands were expressed as the arbitrary units. **P < 0.01 versus the Control group, ##P < 0.01 versus the 100 μM OA group.