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No role for COX(cyclooxygenase)–derived prostanoids in endothelium‐dependent relaxation in response to acetylcholine. Small mesenteric arteries from male and female EC‐MR–intact mice (EC‐MR+/+; closed symbols) and knockout mice (EC‐MR−/−; open symbols) were evaluated in the presence of the COX inhibitor indomethacin (Indo) or vehicle (Veh). Two‐factor ANOVA, P>0.05. EC‐MR indicates mineralocorticoid receptors in endothelial cells.
