Fig. 7.

Model of OsCPK10 function in blast disease resistance and drought tolerance. Biotic and abiotic stresses, such as pathogens or drought stress, induce calcium signals that activate plasma membrane-associated OsCPK10. Catalase A protein interacts with OsCPK10, which could activate its enzymatic function by phosphorylation, resulting in H₂O₂ detoxification, a reduction in lipid peroxidation and its consequent membrane damage. This protection from oxidative damage mediates blast disease resistance and drought tolerance.