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. 2017 Jun 22;68(13):3673–3688. doi: 10.1093/jxb/erx199

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© The Author 2017. Published by Oxford University Press on behalf of the Society for Experimental Biology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 12. — TEM micrographs of sieve elements in different infected A. thaliana lines. Different A. thaliana lines were indicated as follows: (A) wild-type, (B) Atseor1ko, (C) Atseor2ko, (D) Atseor1ko/Atseor2kd, (E) Atseor1kd/Atseor2ko, and (F) Atpp2-a1ko. In infected tissue, wild-type (A) and Atpp2-a1ko (F) SEs showed a massive presence of SE protein filaments (arrows, insets). Also in A. thaliana lines lacking one or both AtSEOR genes (B–E), infected SEs showed filaments with morphology and organization similar to those observed in wild-type plants (arrows, insets). For each condition, five non-serial sections from 10 different plants were analysed. CC, companion cell; m, mitochondrion; ph, phytoplasma; SE, sieve element. Scale bars: 1 µm.