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. 2018 Jan 29;470(4):633–648. doi: 10.1007/s00424-018-2112-5

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© The Author(s) 2018

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.

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Fig. 4 — TRPV4-dependent vasodilation in gracilis arteries. Representative traces of GSK101-induced vasodilation in endothelium intact (gray lines) and denuded (black lines) gracilis arteries from normoxic rats [a]. Summary of the GSK1016790A (GSK101)-induced dilation under endothelium intact and denuded conditions [b]. GSK101 elicits a concentration-dependent vasodilation in arteries from normoxic and CH rats. TRPV4-dependent vasodilation was inhibited by disruption of the endothelium in arteries from both groups. n = 5/group. *Different from endothelium intact within groups (P < 0.05). ^#Different from 10 nM GSK101 within groups (P < 0.05)