Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2018 Jan 30;19(2):395. doi: 10.3390/ijms19020395

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2018 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

The cancer-fighting mechanisms of BP. The effects of BP in brain cancer therapy include impacts on cancer cell apoptosis, cell cycle arrest, senescence, and telomerase inactivation. The compound also exhibits a relatively high level of penetration through the blood-brain barrier (BBB). Taken together, these impacts and properties have been shown to be anti-proliferative, anti-drug resistance, anti-invasion, anti-migration, and anti-metastatic. Symbol key: Pathway; Inhibition; Final effect. Please see the content in Section 5.1.

Inline graphic — The cancer-fighting mechanisms of BP. The effects of BP in brain cancer therapy include impacts on cancer cell apoptosis, cell cycle arrest, senescence, and telomerase inactivation. The compound also exhibits a relatively high level of penetration through the blood-brain barrier (BBB). Taken together, these impacts and properties have been shown to be anti-proliferative, anti-drug resistance, anti-invasion, anti-migration, and anti-metastatic. Symbol key: Pathway; Inhibition; Final effect. Please see the content in Section 5.1.