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. 2018 Jan 31;19(2):421. doi: 10.3390/ijms19020421

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© 2018 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Binding of Rituxan^® (A,B) and _NbRTX^A2G2 (C,D) to Wil2-S and Daudi cells analyzed by flow cytometry. Rituxan^® and _NbRTX^A2G2 were used at a concentration of 10 nM. All rituximab samples were measured in triplicates (red, green and blue lines). FITC-labeled mouse IgG2a (black line) and unstained cells (grey line) were used as controls. The X-axis represents the fluorescent signals of FITC whereas the Y-axis presents % of cell count.

Binding of Rituxan^® (A,B) and _NbRTX^A2G2 (C,D) to Wil2-S and Daudi cells analyzed by flow cytometry. Rituxan^® and _NbRTX^A2G2 were used at a concentration of 10 nM. All rituximab samples were measured in triplicates (red, green and blue lines). FITC-labeled mouse IgG2a (black line) and unstained cells (grey line) were used as controls. The X-axis represents the fluorescent signals of FITC whereas the Y-axis presents % of cell count.