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. 2018 Mar 15;15:82. doi: 10.1186/s12974-018-1121-9

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — The long-term effect of STZ-induced hyperglycemia on synaptogenesis on P90. Representative fluorescent photomicrographs of the hippocampal CA1 region immunostained for MAP2 (red) in a control and b hyperglycemia groups. Scale bars = 25 μm. Representative micrographs demonstrate qualitatively disorganized and truncated dendrites in the formerly hyperglycemic hippocampus compared with the hippocampus of the control rat. c The quantified average integrated density of fluorescent staining was decreased by 16% in the hyperglycemia group (gray) relative to the control group (black), *p < 0.05. d Protein expression of PSD95 in the hippocampus on P90 was decreased by 11% in the hyperglycemia group (gray) relative to the control group (black), *p < 0.05