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. 2018 Feb 26;8(2):2045894018764273. doi: 10.1177/2045894018764273

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© The Author(s) 2018

Creative Commons Non Commercial CC BY-NC: This article is distributed under the terms of the Creative Commons Attribution-NonCommercial 4.0 License (http://www.creativecommons.org/licenses/by-nc/4.0/) which permits non-commercial use, reproduction and distribution of the work without further permission provided the original work is attributed as specified on the SAGE and Open Access pages (https://us.sagepub.com/en-us/nam/open-access-at-sage).

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Fig. 2. — Bar graph showing distribution of PH by etiology, expressed as percentage of total number of African Americans (AAs, n = 586) and Caucasians (n = 3990) in our cohort. AAs had higher prevalence of PH (73% vs. 56%), PAH (16% vs. 11%), Cpc-PH (13% vs. 7%), and WHO group 3 PH (6% vs. 4%), but not Ipc-PH (33% vs. 30%). *P < 0.05.