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. 2018 Mar 20;8:4878. doi: 10.1038/s41598-018-23165-0

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Expression of renal injury and pro-inflammatory markers. (A) Renal mRNA levels of lipocalin 2 (Lcn2; also known as neutrophil gelatinase–associated lipocalin [Ngal]), (B) kidney injury molecule 1 (Kim1) and pro–inflammatory cytokine (C) interleukin 1 beta (Il1b) and (D) Il6 and (E) tumor necrosis factor (Tnf) in sham and in Trpv4 KO and WT mice following IRI. Renal gene expression data are determined in n = 4 for sham-operated WT and Trpv4 KO mice and n = 6 for I/R–injured WT and Trpv4 KO mice, two-way ANOVA. In all cases P_Treatment < 0.001, and P_Genotype and P_Interaction are not significant.