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. 2018 Mar 22;16:27. doi: 10.1186/s12951-018-0357-y

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© The Author(s) 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — CLSM images of human endothelial cells exposed to HA particles (10 and 50 μg/mL) and stained for F-Actin cytoskeleton. On control cells, the cytoskeleton was organized in a network of well-defined and filamentous stress fibres; HAp1 had little effect in this behaviour, whereas HAp2 caused dose-dependent disruption in the cytoskeleton that was shown by diffuse F-actin organization, less stress fibre formation and F-actin condensation (particularly with 50 μg/mL)