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. 2018 Jan 15;12(4):981–996. doi: 10.1038/s41396-017-0034-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a Promoters and read coverage of rpoS, Aln1a-c, and csrA genes at 37 and 43 ppt. The potential CsrA-binding sites within the rpoS and csrA 5′-UTRs are highlighted in red. The three sRNAs (Aln1a-c) originate from the 404 nt intergenic region between the genes Te101_04650 and Te101_04655 from three distinct nTSSs. b Multiple sequence alignment and predicted secondary structures of Aln1a-c. Identified TSSs are indicated by curved arrows on top of each sequence block. Conserved base-pairing are connected with arc curves on top of the alignments. Potential CsrA-binding sites are highlighted by green shadows in the right panel