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. 2018 Mar 16;9:226. doi: 10.3389/fphar.2018.00226

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Copyright © 2018 Hou, Chen, Fang, Cai, Tang, Peng, Deng, Cao, Li, Zhang and Yan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The proposed mechanism of TP-induced hepatotoxicity and ISL combined treatment protecting against TP-induced liver injury. ARE, antioxidant responsive element; BA, bile acid; BSEP, bile salt export pump; conj, conjugated; GSH, reduced glutathione; ISL, isoliquiritigenin; MRP, multidrug resistance protein; NQO1, NAD(P)H: quinine oxidoreductase 1; Nrf2, nuclear transcription factor E2-related factor 2; OATP, organic anion transporting polypeptide; P-gp, P-glycoprotein; sMaf, small Maf proteins; TP, triptolide.