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. 2017 Dec 14;6:401. doi: 10.1038/s41389-017-0003-9

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© The Author(s) 2017

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — High levels of MKP1 correlate with extended glioblastoma patient survival. a MKP1 mRNA levels were assayed in a set of healthy brain tissues as control (n = 6), glioma cell lines (U87MG, U373MG, U251, A172, and T98), and patient-derived GSC cell lines (GNS166, GNS179, GB2, and GB1). qRT-PCR data are normalized to GAPDH expression. b MKP1 mRNA levels were assayed in a cohort including a set of healthy brain tissue as control (n = 6) and glioma samples (n = 59, 44 of them were GBM). qRT-PCR data are normalized to GAPDH expression and expression in tumors is relative to healthy brain tissue. c Analysis of the correlation of MKP1 expression with different glioma grade samples in two independent cohorts. d Kaplan–Meier curve representing the survival of the GBM patients relative to their MKP1 expression levels (n = 44). e Kaplan–Meier curve representing the survival of the GBM patients relative to their MKP1 expression levels (n = 25)