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. 2017 Aug 22;16(5):5915–5923. doi: 10.3892/mmr.2017.7327

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Copyright: © Dong et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 6. — Knockdown of RBP4 altered the expression of related regulators and signaling pathways in HEPM cells. (A) Protein levels of RBP4, cyclin D1, PCNA, p-ERK1/2 and p-AKT were all downregulated in HEPM cells treated with RBP4 siRNA, however p27 was upregulated compared with the Con group. (B) Data for the protein levels are presented as the mean + standard deviation. A total of 3 duplicate samples were used in each group, and experiments were performed in triplicate. **P<0.01 and ***P<0.001. RBP4, retinol binding protein 4; HEPM, human embryonic palatal mesenchymal; PCNA, proliferating cell nuclear antigen; p-, phosphorylated; ERK, extracellular signal-related kinase; AKT, protein kinase B; siRNA, small interfering RNA; Con, control siRNA.