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. 2017 Sep 12;16(5):6715–6721. doi: 10.3892/mmr.2017.7469

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Copyright: © Ding et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Effect of GSKI on the histopathology of LPS-induced lung injury and survival over 14 days. Histopathological analysis of lung tissues from mice in the (A) control, (B) ARDS and (C) ARDS+GSKI groups were performed at indicated times following LPS challenge (hematoxylin and eosin staining; magnification, ×200) (D) Quantification of lung injury showed a significant reduction in the severity of lung injury in the LPS+GSKI group. The change was more marked in the LPS group, compared with that in the LPS+GSKI group. All values are presented as the mean ± standard deviation (n=6–8 animals). *P<0.05. GSKI, glycogen synthase kinase-3β inhibitor; LPS, lipopolysaccharide; ARDS, acute respiratory distress syndrome.