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. 2017 Aug 23;16(5):5997–6003. doi: 10.3892/mmr.2017.7341

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Copyright: © Feng et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — (A) Western blotting and (B) semi-quantitative analysis indicating that levels of GPR30 were significantly increased in HTR8/SVneo cells that had been treated with E2 or G1 in normal conditions. The increased levels of GPR30 induced by E2 were inhibited by G15. (C) Western blotting and (D) semi-quantitative analysis indicated that levels of GPR30 were significantly increased in HTR8/SVneo cells that had been treated with E2 or G1 under conditions of H/R. The increased levels of GPR30 induced by E2 were inhibited by G15. GPR30, G-protein coupled receptor 30; E2, 17β-estrogen; H/R, hypoxia/reoxygenation; DMSO, dimethyl sulfoxide.