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. 2018 Feb 1;17(4):4899–4908. doi: 10.3892/mmr.2018.8535

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Copyright: © Zhang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 1. — MTA2 expression and regulatory effects on NSCLC cells. (A) MTA2 expression in NSCLC and normal human lung cells. (B) Detection of MTA2 expression in lentivirus-mediated si-MTA2 and MTA2 overexpression NSCLC cells. (C) Analysis of the association between MTA2 and the growth of NSCLC cells. (D) Analysis of the association between MTA2 and the migration of NSCLC cells. Magnification, ×400. (E) Analysis of the association between MTA2 and the invasion of NSCLC cells. Magnification, ×400. (F) Apoptosis rate of A549 cells in conditions of MTA2 overexpression and loss. The data are presented as the mean ± standard error of the mean of three independent experiments. **P<0.01 vs. control. MTA2, metastasis associated protein MTA2; NSCLC, non-small cell lung carcinoma; si, small interfering.