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. 2018 Jan 25;17(4):5125–5131. doi: 10.3892/mmr.2018.8485

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Copyright: © Shi et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — HMGB1 has no significant effect on viability of HM cells. (A) Cells were transfected with different concentrations of HMGB1 for the indicated time periods. Cell viability was determined using MTT assay. (B) Comparison of cell viability in each group of Blank, NC, HM, CL-Model, CL-HM and CL-HM-KD. Data from three independent experiments are presented as the mean ± standard deviation (n=3). HMGB1, high-mobility group box-1; Blank, blank group; NC, negative control group; HM, HMGB1; CL-Model, cariaria lactone induced epilepsy cell model; CL-HM, cariaria lactone induced epilepsy cell model plus HMGB1; and CL-HM-KD, cariaria lactone induced epilepsy cell model plus HMGB1 siRNA; OD, optical density.