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. 2017 Oct 3;314(2):E152–E164. doi: 10.1152/ajpendo.00142.2017

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Fig. 3. — IMTG concentration (A), fractional synthesis rate (B), relationship between IMTG FSR and insulin sensitivity (C), and percentage of whole body palmitate turnover trafficked into IMTG (D) during rest, exercise, and recovery in obese volunteers, individuals with type 2 diabetes, and endurance-trained athletes. The relationship between IMTG synthesis rate and C18:0 ceramide content (E), C18:0 glucosylceramide content (F), and cytosolic DAG content (G) are also shown. #Significantly different from athletes, P < 0.05; *significantly different from T2D, P < 0.05; §significantly different from obese, P < 0.05; ¥significantly different from rest, P < 0.05. Values are means ± SE.

Fig. 3. — IMTG concentration (A), fractional synthesis rate (B), relationship between IMTG FSR and insulin sensitivity (C), and percentage of whole body palmitate turnover trafficked into IMTG (D) during rest, exercise, and recovery in obese volunteers, individuals with type 2 diabetes, and endurance-trained athletes. The relationship between IMTG synthesis rate and C18:0 ceramide content (E), C18:0 glucosylceramide content (F), and cytosolic DAG content (G) are also shown. #Significantly different from athletes, P < 0.05; *significantly different from T2D, P < 0.05; §significantly different from obese, P < 0.05; ¥significantly different from rest, P < 0.05. Values are means ± SE.