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. 2018 Mar 19;9:355. doi: 10.3389/fpls.2018.00355

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Copyright © 2018 Ramirez-Prado, Piquerez, Bendahmane, Hirt, Raynaud and Benhamed.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Nitric oxide (NO) induces HDAC inhibition in a SA-dependent fashion. Increased SA (salicylic acid) levels induce NO biosynthesis and the consequent inhibition of histone deacetylases (HDACs), including HDA19. Such inhibition leads to increased H3 and H4 acetylation levels on stress-responsive genes, inducing their transcriptional activation.