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. 2018 Mar 14;19(3):853. doi: 10.3390/ijms19030853

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© 2018 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (http://creativecommons.org/licenses/by/4.0/).

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Developmental impairment of meristem proliferation and stem cell identity in plants lacking the mitochondrial protease AtFTSH4. In stress-inducing conditions of mildly elevated temperature of 30 °C, absence of AtFTSH4 in mitochondria (shown as dashed X) results in progressive accumulation of internal oxidative stress (ROS, carbonylated proteins, AOX transcripts) with time/plant age and ATP deficiency [22,24,25]. Stem cells within the meristems lose their meristematic characteristics and ability to proliferate. In the shoot apical meristem (SAM), this relates to the transcript dysregulation of cell cycle-related genes and those sustaining meristematic identity. Ultimately, growth of the SAM and RAM in the ftsh4-1 mutant plants is precociously arrested. Question marks refer to probable, but not yet proven experimentally correlations.