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. 2018 Mar 12;115(13):3392–3397. doi: 10.1073/pnas.1717815115

Fig. 6.

Fig. 6.

Nr2f2 and En1/2 influence the differentiation of SmoM2-mutant P8 GCPs but not tumor formation. (A) Graphs comparing the proportions of undifferentiated GCPs between H and V of A-M-SmoM2 (also shown in Fig. 3A), A-M-SmoM2-N (n = 4), and A-M-SmoM2-N het (n = 5) mice. (B) Graphs comparing the proportions of undifferentiated GCPs between the H and V of A-M-SmoM2 and A-M-SmoM2-E (n = 3) mice. (C) Graphs showing the size of H lesions from A-M-SmoM2 (n = 11), A-M-SmoM2-N (n = 12), and A-M-SmoM2-N het (n = 13). Significances determined using a two-way ANOVA followed by a Sidak post hoc test for A and B, or with a one-way ANOVA followed by Tukey post hoc test for C. All data are expressed as mean ± SEM. ****P < 0.0001, **P < 0.01, *P < 0.05, ns, nonsignificant. Statistics are provided in Table S2.