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. 2018 Apr 2;13(4):e0195211. doi: 10.1371/journal.pone.0195211

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© 2018 Croft et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 5 — Immunoblots of biochemically sarkosyl-insoluble temporal cortex lysates from post-mortem control (n = 2) and AD (n = 3) cases were probed with the novel tau antibodies (as indicated for each blot) 81A11, 83E4, 94-3A2, 94-3A6, 94-4F1 to determine affinity for tau in human AD. The mobilities of molecular mass markers are shown on the left.