Fig 7. A model illustrates the effect of FIN219 levels on cry1 functions.

(A) In the dark, FIN219 interacts with the C terminus of CRY1 (CCT1) to likely abolish CRY1 functions. Meanwhile, the COP1–SPA1 complex interacts with HY5, thereby degrading HY5 protein for skotomorphogenic development of Arabidopsis seedlings. (B) In blue light, FIN219 interacts closely with CRY1 as well as COP1, resulting in cry1-mediated photomorphogenesis. (C) In the presence of blue light, induction of FIN219 by MeJA strongly interacts with CCT1, and weakly with COP1–SPA1 complex, leading to a rescued phenotype of GUS-CCT1 seedlings by suppressing hypersensitive short-hypocotyl phenotype.