Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2018 Feb 24;59(4):730–738. doi: 10.1194/jlr.M081026

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Author’s Choice—Final version open access under the terms of the Creative Commons CC-BY license.

PMC Copyright notice

Fig. 3. — Differential content of cPLA₂ and AA in CEA plaque segments of diabetic subjects. A: Representative Western blot (WB) demonstrates higher cPLA₂ abundance in the MAX and MIN diseased CEA segments of nondiabetic subjects. GAPDH is a loading control between lanes. B: Quantitative Western blot cPLA₂ band intensity, normalized to GAPDH, demonstrated higher cPLA₂ in MIN and MAX diseased segments of nondiabetic subjects (n = 5) compared with diabetic subjects (n = 6). C: Representative Western blot demonstrates no difference in iPLA₂ abundance in the MAX and MIN diseased CEA segments of diabetic and nondiabetic subjects. D: Quantitative Western blot iPLA₂ band intensity, normalized to GAPDH, demonstrates no difference in iPLA₂ in MIN and MAX diseased segments of diabetic (n = 6) and nondiabetic subjects (n = 5). E: ELISA analysis of AA content in MAX and MIN diseased CEA segments demonstrates higher AA content in MIN segments in both diabetic (n = 7) and nondiabetic (n = 5) patients. Error bars = SEM, *P < 0.05 and ***P < 0.001.