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. 2018 Mar 6;41(6):3127–3136. doi: 10.3892/ijmm.2018.3537

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Copyright: © Yang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Generation of ROS in H9C2 cells during the process of cell apoptosis. (A) Compared with in the control group, ROS, as measured using dihydroethidium, was significantly decreased in TP-treated cells in a dose-dependent manner. (B and C) TP exerted protective effects against I/R via SOD, MDA and CAT. ^***P<0.001, compared with the control group. ^##P<0.01, ^###P<0.001, compared with the I/R group (n=3). CAT, catalase; I/R, ischemia/reperfusion; MDA, malondialdehyde; ROS, reactive oxygen species; SOD, superoxide dismutase; TP, triptolide.