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Schisandrin (SIN) treatment improved the escape latency (A) and path length (B) in the navigation test and improved performance of the probe trial in MWM of APP/PS1 mice (n=10). (C) The representive locus plot after SIN treatment in the probe trial. (D) SIN treatment prolonged the time spent in target quadrant. (E) SIN treatment increased the frequencies of passing through the goal. **P<0.01, compared with normal group; ##P<0.01, compared with APP/PS1 group.
