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. 2018 Mar 27;9:272. doi: 10.3389/fphar.2018.00272

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Copyright © 2018 Zeng, Li, Wei, Hu, Jiao, Yu and Xiong.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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EphA2 overexpression abolished the protective effects of ALW-II-41-27 on LPS-induced oxidative stress and inflammation. Effects of EphA2 overexpression on cell viability (A), LDH leakage (B), cellular 4-HNE (C), cellular protein carbonyl (D), cellular 8-OHdG (E), cellular HO-1 activity (F), cellular TNF-α (G), cellular IL-6 (H), cellular IL-17 (I), cellular ICAM-1 (J), cellular IL-10 (K) and cellular NF-κB activity (L). ^##Indicates a significant difference from control cells (P < 0.01); ^∗indicates a significant difference from LPS group (P < 0.05); ^∗∗indicates a significant difference from LPS group (P < 0.01); ^ΔΔindicates a significant difference from control cells treated LPS; ^&&indicates a significant difference from control cells treated with ALW-II-41-27 and LPS.