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. 2018 Mar 28;16(3):e2004444. doi: 10.1371/journal.pbio.2004444

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© 2018 Kennedy, Dwyer

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 3 — Comparison of the predictions of our models (gray-shaded areas, showing 95% CIs of model realizations) to the distribution of nucleotide diversity within 143 individual infected hosts calculated from our sequence data (black dots show data on nucleotide diversity within hosts). Panel A shows the predictions of a model that lacks both transmission bottlenecks and replicative drift, panel B shows the predictions of a model that includes transmission bottlenecks but not replicative drift, panel C shows the predictions of a model that includes both transmission bottlenecks and replicative drift, and panel D shows the predictions of a model that includes purifying selection within hosts but not transmission bottlenecks or replicative drift. Values underlying data points are provided in S1 Data.