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. 2018 Feb 25;109(4):1121–1134. doi: 10.1111/cas.13531

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© 2018 The Authors. Cancer Science published by John Wiley & Sons Australia, Ltd on behalf of Japanese Cancer Association.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

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SIM2 induces well‐differentiated histology and in vivo growth suppression of KYSE510 and T.Tn cells. A, Xenograft tumor growth curves of mock‐ and SIM2‐transfectants of KYSE510 and T.Tn in 3 sites of 2 mice (n = 6, mean + SE). B, Hematoxylin and eosin staining and immunocytochemistry for PDPN in xenografts of KYSE510‐Mock, KYSE510‐SIM2‐27, T.Tn‐Mock, and T.Tn‐SIM2‐9. C, Number of cancer pearls per slice in the xenograft of KYSE510‐Mock, KYSE510‐SIM2‐27, KYSE510‐SIM2‐37, T.Tn‐Mock, T.Tn‐SIM2‐23, and T.Tn‐SIM2‐9. Gray bars represent average number of cancer pearls. Each number of cancer pearls between the Mock‐ and SIM2‐transfectants was discriminated by white and red (n = 6, *P < .05)