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. 2018 Apr 4;9:634. doi: 10.3389/fimmu.2018.00634

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Copyright © 2018 Bejar, Kessler, Sabag, Sabo, Itzhak, Neufeld and Vadasz.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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(A) Hematoxylin–eosin staining shows paucity of blood capillaries and diffuse mesangial proliferation in control-treated mice (left panel). These findings were minimal in semaphorin3A (sema3A)-treated mice (right panel). (B) PAS staining demonstrates profound glycoprotein deposits in the tubuli of control-treated mice (left panel) and minimal deposits in sema3A-treated mice (right panel). (C) Increased glycoprotein deposits are seen in the mesangium of control-treated mice (left panel) and minimal in sema3A-treated mice (right panel).