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. 2018 Apr 11;18:407. doi: 10.1186/s12885-018-4224-x

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — Slc10a2 via PPARγ plays an important role in tumor suppressor with the treatment of bexarotene. a The expression of apoptotic related genes Bcl-2, cyclin D1, c-FLIP, caspase 3, caspase 7 in A549 cells and slc10a2 overexpressed A549 cells when treated with bexarotene, bexarotene in combination with GW9662 respectively; b The expression of tumor suppressor genes PTEN, P21, P53, LKB1, TSC2 in A549 cells and slc10a2 overexpressed A549 cells when treated with bexarotene, bexarotene in combination with GW9662 respectively. A549 cells without any treatment as control group. All experiments were repeated 3 times